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Schematic illustration of the molecular mechanism of GNB4 in H. pylori -induced GC. H. pylori induces GNB4 upregulation via the NF-κB-TET1 axis. TET1-mediated demethylation of GNB4 promoter leads to the transcriptional activation of GNB4, and high GNB4 expression contributes to GC progression via the Hippo–YAP1 pathway

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