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Workflow from Scientific Research

Open access visualization of Workflow, Illustration, Alzheimer's Disease, Microglial Cells, Apolipoprotein E

#Workflow #Illustration #Alzheimer's Disease #Microglial Cells #Apolipoprotein E #TREM2 Signalling #Astrocytes #Oligodendrocytes #Neuroinflammatory State #Immune Cell Infiltration #Amyloid Beta #Blood-Brain Barrier #Complement C1q A Chain #Disease-Associated #Interferon #Interleukin 1A #Serpin Family A Member 3 #Tau #Transforming Growth Factor Beta 1 #Tumor Necrosis Factor

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Illustration Alzheimer's Disease Microglial Cells

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DOI: 10.1038/s41421-024-00666-z

Alzheimer’s disease (AD) develops in the context of complex immunological alterations that involve not only microglial cells, with a major role for altered apolipoprotein E (APOE) and triggering receptor expressed on myeloid cells 2 (TREM2) signalling, but also astrocytes and oligodendrocytes, culminating with a neuroinflammatory state associated with immune cell infiltration from the periphery. A amyloid beta, BBB blood-brain barrier, C1QA complement C1q A chain, DA disease-associated, IFN interferon, IL1A interleukin 1A, SERPINA3 serpin family A member 3, tau (microtubule-associated protein tau, MAPT), TGFB1 transforming growth factor beta 1, TNF tumor necrosis factor. Created with BioRender.com.

DOI

10.1038/s41421-024-00666-z

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